December 30, 2021

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For instance, impaired BMP signaling is important in the progression of colon cancer. More research clearly needs to be done, particularly in animal models, to better quantify how follistatin can be used in the treatment and prevention of BMP misregulation. Follistatin Research and Cancer Treatment. Researchers have found that follistatin active in a number of different tumors and that its effects are not just limited to breast cancer and liver carcinoma. With better understanding of how follistatin works, it is hoped that a number of clinical applications for the protein can be developed. Currently, follistatin expression has been associated with improved survival in breast cancer, but shortened survival in lung, ovarian, and gastric cancers. It is hoped that understanding these differences in how the protein affects survival will allow for the development of treatment adjuvant tailored to specific types of cancer. Follistatin research may even form the basis of a cancer preventative or vaccine, slowing rates of metastasis and improving long-term survival [9]. Follistatin Associated with Cell Proliferation. The research into breast cancer revealed an odd dichotomy in that follistatin promotes cell proliferation but restricts metastasis. As it turns out, this dichotomy of follistatin is true in most tissues. In the liver, in particular, hepatocytes require follistatin expression in order to proliferate. Rat models show that the inactivation of activin via follistatin may be necessary for proliferation to take place [10]. This may explain why follistatin is associated with increased tumor growth, but decreased tumor invasion and metastasis. There is likely an energy trade off that takes place during growth in which the migratory functions of a cell are shut off in order to shunt energy into growth and reproduction. Follistatin Research and Liver Protection. Follistatin has been shown to protect the liver against the earliest phases of fibrosis and thus slow progression of liver disease. Research in rats shows a 32% reduction in fibrosis after four weeks of follistatin treatment, a positive trait attributed to an 87% reduction in hepatocyte death in treatment groups [11]>. Dysregulation of follistatin has been linked to the progression of fibrosis to cancer in the liver [12]. Follistatin Provides Insight into Congenital Blindness. The ability of the optic nerve to fuse in early development is critical to ability of humans to see. It has been known for some time that TGF-beta signaling is critical to remodeling of the extracellular matrix and eventual fusion of the optic nerve. Research suggests that high levels of TGF-beta proteins, particularly bone morphogenic protein (BMP), suppress optic fusion and can lead to blindness. 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