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Cabaser tablets for oral administration contain 1. Excipient(s) with known effect. Adjunctive therapy to levodopa – treatment of motor complications. The pharmacokinetic and metabolic profiles of Cabaser have been studied in healthy volunteers of both sexes, in female hyperprolactinaemic patients and in parkinsonian patients. After oral administration of the labelled compound, radioactivity was rapidly absorbed from the gastrointestinal tract as the peak of radioactivity in plasma was between 0. Ten days after administration about 20% and 72% of the radioactive dose ( 14 C-cabergoline) was recovered in urine and faeces, respectively. Unchanged drug in urine accounted for 2-3% of the dose. In urine, the main metabolite identified was 6-allyl-8b-carboxy-ergoline, which accounted for 4-6% of the dose. Three additional metabolites were identified in urine which accounted overall for less than 3% of the dose. The metabolites have been found to be much less potent than Cabaser as D 2 -dopamine receptor agonists in vitro. The low urinary excretion of unchanged Cabaser has also been confirmed in studies with nonradioactive product. The elimination half-life of Cabaser, estimated from urinary excretion rates, is long (63-68 hours in healthy volunteers, 79 to 115 hours in hyperprolactinaemic patients). The pharmacokinetics of Cabaser seem to be linear both in healthy volunteers (doses of 0. On the basis of the elimination half-life, steady state conditions should be achieved after 4 weeks, as confirmed by the mean peak plasma levels of Cabaser obtained after a single dose (37 ‘ 8 picogram/mL) and after a 4 week multiple regimen (101 ‘ 43 picogram/mL). In vitro experiments showed that the drug at concentrations of 0. In limited studies, food did not appear to have an effect on the absorption and disposition of Cabaser. Renal insufficiency has been shown not to modify cabergoline kinetics. Hepatic insufficiency with a Child-Pugh score of less than 10 does not appear to affect the pharmacokinetics of cabergoline. A single study in which 12 patients received 1 mg cabergoline as a single dose, showed that there was no statistical difference in Cmax, Tmax and AUC in patients with mild liver disease group (group A, average Child-Pugh score 5. It was observed however, that the average AUC tended to increase with the severity of the hepatic impairment, particularly in group C. For the management of the signs and symptoms of Parkinson’s disease. Treatment should be initiated under specialist supervision. The benefit of continued treatment should be regularly reassessed taking into account the risk of fibrotic reactions and valvulopathy (see Section 4. Hypersensitivity to cabergoline, other ergot alkaloids or to any of the excipients, buy test-r 200 mg injectable steroids sustanon (testosterone blend). History of pulmonary, pericardial or retroperitoneal fibrotic disorders.
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