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The main difference between androgenic and anabolic is that androgenic steroids generate male sex hormone-related activity whereas anabolic steroids increase both muscle mass and the bone mass. However, despite the similarities in structure, function, and usage, androgenic and anabolic steroids behave very differently. There have been some attempts by androgenic steroids to mimic their anabolic counterparts, mainly for the purpose of weight-loss purposes or weight gain control. The main androgenic steroid is testosterone, which is synthesized from the seminal vesicles and is a potent androgen, with comparable androgenic strength, but is also produced only in excess, and has a higher affinity for the liver for this conversion which, in turn, also leads to a higher rate of androgen production, which leads to higher gains of weight.
While the metabolism of testosterone varies, the metabolic pathways for its production are similar, i.e. through the liver, through the pituitary, and then on to the muscle cells, where they undergo conversion into the steroid hormone dihydrotestosterone (DHT). Dihydrotestosterone is then converted into testosterone, which acts as a muscle relaxant. While testosterone is known to stimulate androgen secretion during androgen deprivation and muscle growth, this is most likely the dominant androgenic steroid action in the muscle, and is most likely mediated by the steroid receptors (androgen receptors such as ER and FSH) on the muscle cell surface. In an in vitro study, human androgen receptor (AR) α protein levels were increased significantly during stimulation of the sympathetic nervous system (SNS) (stimulating AR) and ARβα/ARβα kinase activity as well as increasing levels of and AR phosphorylation in muscle tissue following an experimental training regimen of a resistance training, non-competitive bout. The increase in α mRNA levels was also associated with increases in the activity of the androgen receptor on muscle fibre membranes. This suggests, it may also be one mechanism through which an androgenic steroid might potentially increase the size of muscles.
It has been hypothesized that the androgenic steroids have a similar metabolic profile in the muscle due to the fact that the androgen-secreting hormone is released through transmembrane release from the androgen receptor on the membrane of the muscle tissue and via a trans-membrane release pathway from the muscle cell receptors. Furthermore, the androgen receptor also acts in vitro to stimulate the activity of the muscle transenzyme to convert from dihydrotestosterone to the androgenic steroid and then to testosterone when trans-transport is initiated to
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